glutathione mitochondrial disease Depletion in Diseases Mitochondrial Glutathione: Regulation and Functions

*Correspondence: Silke Keiner, [email protected] This article was submitted to Neurogenesis, a section of the journal Frontiers in Neuroscience Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

1 Introduction Sepsis-associated encephalopathy (SAE) represents a severe complication of sepsis, defined by diffuse brain dysfunction without direct central nervous system (CNS) infection ( Oxidative stress is central to SAE pathogenesis, driven by an imbalance between excessive reactive oxygen species (ROS) production and impaired antioxidant defense systems ( The antioxidant defense system, especially GSH metabolism, is linked to neurodegenerative diseases ( Proteomics and metabolomics enable systematic profiling of protein expression and metabolic signatures

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Patients at MOPE Clinic in Metairie ask about it regularly, especially those already on GLP-1 therapy who want to understand whats coming next